Category: Elisa Kits

Allogeneic hepatocyte transplantation: Contribution of Fas-Fas ligand interaction to allogeneic hepatocyte rejection.

Hepatocyte transplantation is a possible therapeutic modality for overcoming the scarcity of liver donors, and the scientific software of allogeneic hepatocyte transplantation has been thought-about. Nevertheless, there are two main issues with allogeneic hepatocyte transplantation: safety of transplanted hepatocytes from rejection and stimulation of the fast proliferation of surviving cells. With out immunosuppression, allogeneic hepatocytes

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Frequency of a FAS ligand gene variant associated with inherited feline autoimmune lymphoproliferative syndrome in British shorthair cats in New Zealand.

AIMS To find out the frequency of the FAS-ligand gene (FASLG) variant related to feline autoimmune lymphoproliferative syndrome (FALPS) and the proportion of carriers of the variant in three British shorthair (BSH) breeding catteries in New Zealand. METHODS Buccal swabs have been collected from all cats in two BSH breeding catteries from the South Island

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Human intrahepatic regulatory T cells are functional, require IL-2 from effector cells for survival, and are susceptible to Fas ligand-mediated apoptosis.

Regulatory T cells (Treg ) suppress T effector cell proliferation and preserve immune homeostasis. Autoimmune liver illnesses persist regardless of excessive frequencies of Treg within the liver, suggesting that the native hepatic microenvironment may have an effect on Treg stability, survival, and performance. We hypothesized that interactions between Treg and endothelial cells throughout recruitment after

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Dephosphorylation of Fas-ligand and caveolin-1 is a prerequisite step in Fas-ligand – caveolin-1 complex formation and cell death stimulation.

Fas-ligand/CD178 belongs to the TNF household proteins and is the well-characterized inducer of cell demise. We confirmed beforehand that the interplay of Fas-ligand and caveolin-1 is important for Fas-ligand translocation to rafts, and the following induction of Fas-ligand-dependent cell demise. Each molecules can endure phosphorylation, nevertheless the function of the phosphorylation state of Fas-ligand and

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